RVA-induced necrosome formation consisting of RIPK1, RIPK3, and MLKL
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Post-translational control of RIPK3 and MLKL
MLKL in cancer: more than a necroptosis regulator. - Abstract - Europe PMC
Biomolecules, Free Full-Text
RIP3 is an upregulator of aerobic metabolism and the enhanced respiration by necrosomal RIP3 feeds back on necrosome to promote necroptosis
RIP1/RIP3-regulated necroptosis as a target for multifaceted disease therapy (Review)
RIPK3-and MLKL-dependent necroptosis mediated by various stimuli.
The Structural Basis of Necroptotic Cell Death Signaling: Trends in Biochemical Sciences
RIPK3 Activates Parallel Pathways of MLKL-Driven Necroptosis and FADD-Mediated Apoptosis to Protect against Influenza A Virus - ScienceDirect
Targeting Necroptosis as a Promising Therapy for Alzheimer's Disease
Human RIPK3 maintains MLKL in an inactive conformation prior to cell death by necroptosis
Frontiers Exploiting Necroptosis for Therapy of Acute Lymphoblastic Leukemia
Frontiers Up-Regulation of RIP3 Alleviates Prostate Cancer Progression by Activation of RIP3/MLKL Signaling Pathway and Induction of Necroptosis
Bypassing drug resistance by triggering necroptosis: recent advances in mechanisms and its therapeutic exploitation in leukemia, Journal of Experimental & Clinical Cancer Research
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