Frontiers BETting on a Transcriptional Deficit as the Main Cause for Cornelia de Lange Syndrome
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Down syndrome-like acute megakaryoblastic leukemia in a patient with Cornelia de Lange syndrome
Lyar-Mediated Recruitment of Brd2 to the Chromatin Attenuates Nanog Downregulation Following Induction of Differentiation - ScienceDirect
Frontiers BETting on a Transcriptional Deficit as the Main Cause for Cornelia de Lange Syndrome
STAG2 promotes the myelination transcriptional program in oligodendrocytes
Cornelia de Lange syndrome mutations in NIPBL can impair cohesin-mediated DNA loop extrusion
Functional coordination of BET family proteins underlies altered transcription associated with memory impairment in fragile X syndrome
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Diagnostic pathways for Cornelia de Lange (CdLS) Syndrome. In
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Cornelia de Lange Syndrome (CdLS) is a cohesinopathy (changes in
Neuronal genes deregulated in Cornelia de Lange Syndrome respond to removal and re-expression of cohesin
STAG2 promotes the myelination transcriptional program in oligodendrocytes
Functional coordination of BET family proteins underlies altered transcription associated with memory impairment in fragile X syndrome
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